So excited and grateful to have been named a 2025 Leading Edge Fellow and for the opportunity to be a part of this amazing community!

Leading Edge Fellows Program@leadingedgeprogram.bsky.social · last yr.

We are thrilled to announce the 2025 Leading Edge Fellows! 40 outstanding postdoctoral fellows doing pioneering research in a wide range of biological and biomedical disciplines. Learn more about these exceptional scientists: www.leadingedgesymposium.org/fellows

UDS members enjoying the 14th International BMP conference in Philly 😁

@jimmykjm.bsky.social and @isabellaclsci.bsky.social et al. focus on epigenetic changes during fin regeneration in this preprint: www.biorxiv.org/content/10.1...

Early epigenetic priming of regeneration studied with a novel transgenic epigenetic reporter

Tissue regeneration requires previously differentiated cells to regain developmental plasticity. However, the upstream mechanisms initiating this process remain poorly understood. Here, we leverage a ...

biorxiv.org

So excited to share the first preprint of my postdoc work with @katwrog.bsky.social! 💡🐟

Katherine W. Rogers@katwrog.bsky.social · last yr.

Happy to share a new preprint from my lab! We characterize the on/off kinetics, light dosage-dependence, and more for a suite of optogenetic signaling activators in zebrafish embryos 💡 www.biorxiv.org/content/10.1...

“Together, our results establish this optogenetic toolkit as a potent experimental platform to rapidly, directly, and adjustably activate FGF, BMP, and Nodal signaling in zebrafish embryos.” New work from @leanneiannucci.bsky.social & @katwrog.bsky.social 🔥

bioRxiv Developmental Biology@biorxiv-devbio.bsky.social · last yr.

An optogenetic toolkit for robust activation of FGF, BMP, and Nodal signaling in zebrafish https://www.biorxiv.org/content/10.1101/2025.04.17.649426v1

I am thrilled to share our recent publication, led by recent PhD graduate Dr. Syeda Lamia + collab with @brianglancylab.bsky.social, on how overexpression of EYFP using optogenetic mouse strains leads to skeletal muscle contractile dysfunction. 🧵 faseb.onlinelibrary.wiley.com/doi/full/10....

Overexpression of enhanced yellow fluorescent protein fused with Channelrhodopsin‐2 causes contractile dysfunction in skeletal muscle

Optogenetic tools like Channelrhodpsin-2 (ChR2) are useful for inducing skeletal muscle contraction using blue light. However, the prevalence of fluorescent proteins like EYFP may cause functional is...

faseb.onlinelibrary.wiley.com